Document Type

Article

Publication Date

7-22-2021

Publication Title

Cell Mol Gastroenterol Hepatol

Abstract

Pancreatic ductal adenocarcinoma (PDA), the most common pancreatic cancer, is a nearly-universally lethal malignancy. PDA is characterized by extensive infiltration of immunosuppressive myeloid cells, including tumor-associated macrophages (TAMs) and myeloid-derived suppressor cells (MDSCs). Myeloid cells in the tumor microenvironment (TME) inhibit cytotoxic T cell responses promoting carcinogenesis. Immune checkpoint therapy has not been effective in PDA, most likely due to this robust immune suppression, making it critical to elucidate mechanisms behind this phenomenon. Here, we review myeloid cell infiltration and cellular crosstalk in PDA progression and highlight current therapeutic approaches to target myeloid cell-driven immune suppression.

Medical Subject Headings

Animals; Biomarkers; Cell Communication; Cell Lineage/immunology; Cell Transformation; Neoplastic/genetics/immunology/metabolism; Disease Management; Disease Susceptibility; Drug Resistance; Neoplasm; Gene Expression Profiling; Gene Expression Regulation; Neoplastic; Humans; Immunomodulation; Myeloid Cells/immunology/metabolism; Myeloid-Derived Suppressor Cells/immunology/metabolism/pathology; Neoplasm Metastasis; Pancreatic Neoplasms/etiology/metabolism/pathology/therapy; Signal Transduction; Single-Cell Analysis/methods; Tumor Microenvironment/immunology; Tumor-Associated Macrophages/immunology/metabolism/pathology

PubMed ID

34303882

ePublication

ePub ahead of print

Volume

12

Issue

5

First Page

1531

Last Page

1542

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